Quick Detail

Product name: Liothyronine Sodium (T3)

Alias: Cytomel T3; 3, 3′, 5-triiodothyronine sodium;T3

Einecs No: 200-223-5

MF: C15H11I3NNaO4

CAS No: 55-06-1

MW: 672.96

Purity: 99.0%

Appearance: white to beige powder

Usage: These hormones also regulate protein, fat, and carbohydrate metabolism, affecting how human cells use energetic compounds.

T3 Dosage

T3 does speed fat loss. As a guideline, for most 12.5 mcg/day is a conservative “supplement” sort of dosing that seems to have no detectable adverse effect on thyroid function at all.

25 mcg/day is a “supplement” sort of dosing that does have some inhibitory effect.

50 mcg/day is a reasonably conservative bb’ing sort of dose that, of course, is more inhibitory.

75 mcg/day is getting into more of a problem area;

100 mcg/day in many cases leads to loss of muscle size and strength.

In this way ,Liothyronine sodium, the goal is to achieve an ongoing edge in fat loss or to help

maintain a near-personally-ideal body composition . In this approach , T3 dosing is very low ,

preferably 12.5 mcg/day but in some cases as much as 25 mcg/day. At the lower end of this range , typically thyroid testing will show no detectable suppression even with prolonged use . At the higher end , moderate suppression is sometimes seen , but results are superior to when T3 is not taken , and the suppression reverses rapidly upon discontinuing T3 use .

In the other way ,Liothyronine sodium, the goal is to achieve a quite substantial increase in rate of fat loss , at the known cost of inducing thyroid suppression . Most preferably the dosage is about 50 mcg/day , but in some instances can be as high as 75 mcg / day . Such use is preferably not ongoing, but only for a limited period of time such as 8-12 weeks , though there’s no exact requirement for timeframe .

T3 VS T4

T4 is deiodinated by three deiodinase enzymes to produce the more-active triiodothyronine.T3 is the more metabolically active hormone produced from T4.

Deiodinase enzyme one accounts for 80% of the deiodination of T4, present in liver, kidney, thyroid, and (to a lesser extent) pituitary.

Deiodinase enzyme two presents in CNS, pituitary, mediates negative feedback on thyroid-stimulating hormone, also presents in brown adipose tissue, and heart vessel, which is predominantly intracellular.

Deiodinase enzyme three, converts T4 into reverse T3, which, unlike T3, is inactive, it present in placenta, CNS, and hemangioma. 

T3 COA